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Several studies report a decrease in GSTP1 in HCC tissues when compared to healthy or HBV patients [317], probably due to the hypermethylation of its promoter [318]

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As both an endogenous antioxidant and a regulator of mitochondrial function, melatonin uniquely bridges circadian regulation and redox homeostasis, positioning oxidative stress as a downstream consequence of circadian and melatonergic decline in aging